Secondary paroxysmal dyskinesias
Identifieur interne : 004296 ( Main/Exploration ); précédent : 004295; suivant : 004297Secondary paroxysmal dyskinesias
Auteurs : Jaishri Blakeley [États-Unis] ; Joseph Jankovic [États-Unis]Source :
- Movement Disorders [ 0885-3185 ] ; 2002-07.
Descripteurs français
- Pascal (Inist)
- Wicri :
- topic : Homme.
English descriptors
- KwdEn :
- Adolescent, Adult, Age of onset, Aged, Bibliographic review, Child, Child, Preschool, Chorea (diagnosis), Chorea (etiology), Diagnosis, Differential, Dyskinesia, Etiology, Female, Human, Humans, Male, Middle Aged, Neurologic Examination, Retrospective Studies, Risk Factors, Secondary, Sudden, Symptomatology, Time lag, chorea, dystonia, paroxysmal dyskinesias, secondary, symptomatic.
- MESH :
- diagnosis : Chorea.
- etiology : Chorea.
- Adolescent, Adult, Aged, Child, Child, Preschool, Diagnosis, Differential, Female, Humans, Male, Middle Aged, Neurologic Examination, Retrospective Studies, Risk Factors.
Abstract
Paroxysmal dyskinesias (PxDs) are involuntary, episodic movements that include paroxysmal kinesigenic (PKD), paroxysmal nonkinesigenic (PNKD), and paroxysmal hypnogenic (PHD) varieties. Although most PxDs are primary (idiopathic or genetic), we found 17 of our 76 patients with PxD (22%) to have an identifiable cause for their PxD (10 men; mean age, 41.4 years). Causes included peripheral trauma (in three patients), vascular lesions (in four), central trauma (in four), kernicterus (in two), multiple sclerosis (in one), cytomegalovirus encephalitis (in one), meningovascular syphilis (in one), and migraine (in one). The latency from insult to symptom onset ranged from days (trauma) to 18 years (kernicterus), with a mean of 3 years. Nine patients had PNKD, two had PKD, five had mixed PKD/PNKD, and one had PHD. Hemidystonia was the most common expression of the paroxysmal movement disorder, present in 11 patients. Both of the patients with PKD had symptom durations of <5 minutes. Symptom duration ranged from 10 seconds to 15 days for PNKD and from 5 minutes to 45 minutes for mixed PKD/PNKD. There were no uniformly effective therapies, but anticonvulsant drugs, clonazepam, and botulinum toxin injections were the most beneficial. Awareness of the variable phenomenology and the spectrum of causes associated with secondary PxD will allow for more timely diagnosis and early intervention. © 2002 Movement Disorder Society
Url:
DOI: 10.1002/mds.10178
Affiliations:
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Le document en format XML
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Disord.</title><idno type="ISSN">0885-3185</idno><idno type="eISSN">1531-8257</idno><imprint><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>New York</pubPlace><date type="published" when="2002-07">2002-07</date><biblScope unit="vol">17</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="726">726</biblScope><biblScope unit="page" to="734">734</biblScope></imprint><idno type="ISSN">0885-3185</idno></series><idno type="istex">18B1C50AC88B370598D43C2AD05219E2CFC13490</idno><idno type="DOI">10.1002/mds.10178</idno><idno type="ArticleID">MDS10178</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0885-3185</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Adolescent</term><term>Adult</term><term>Age of onset</term><term>Aged</term><term>Bibliographic review</term><term>Child</term><term>Child, Preschool</term><term>Chorea (diagnosis)</term><term>Chorea (etiology)</term><term>Diagnosis, Differential</term><term>Dyskinesia</term><term>Etiology</term><term>Female</term><term>Human</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Neurologic Examination</term><term>Retrospective Studies</term><term>Risk Factors</term><term>Secondary</term><term>Sudden</term><term>Symptomatology</term><term>Time lag</term><term>chorea</term><term>dystonia</term><term>paroxysmal dyskinesias</term><term>secondary</term><term>symptomatic</term></keywords><keywords scheme="MESH" qualifier="diagnosis" xml:lang="en"><term>Chorea</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Chorea</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Adolescent</term><term>Adult</term><term>Aged</term><term>Child</term><term>Child, Preschool</term><term>Diagnosis, Differential</term><term>Female</term><term>Humans</term><term>Male</term><term>Middle Aged</term><term>Neurologic Examination</term><term>Retrospective Studies</term><term>Risk Factors</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Age apparition</term><term>Brutal</term><term>Dyskinésie</term><term>Délai</term><term>Etiologie</term><term>Homme</term><term>Revue bibliographique</term><term>Secondaire</term><term>Symptomatologie</term></keywords><keywords scheme="Wicri" type="topic" xml:lang="fr"><term>Homme</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Paroxysmal dyskinesias (PxDs) are involuntary, episodic movements that include paroxysmal kinesigenic (PKD), paroxysmal nonkinesigenic (PNKD), and paroxysmal hypnogenic (PHD) varieties. Although most PxDs are primary (idiopathic or genetic), we found 17 of our 76 patients with PxD (22%) to have an identifiable cause for their PxD (10 men; mean age, 41.4 years). Causes included peripheral trauma (in three patients), vascular lesions (in four), central trauma (in four), kernicterus (in two), multiple sclerosis (in one), cytomegalovirus encephalitis (in one), meningovascular syphilis (in one), and migraine (in one). The latency from insult to symptom onset ranged from days (trauma) to 18 years (kernicterus), with a mean of 3 years. Nine patients had PNKD, two had PKD, five had mixed PKD/PNKD, and one had PHD. Hemidystonia was the most common expression of the paroxysmal movement disorder, present in 11 patients. Both of the patients with PKD had symptom durations of <5 minutes. Symptom duration ranged from 10 seconds to 15 days for PNKD and from 5 minutes to 45 minutes for mixed PKD/PNKD. There were no uniformly effective therapies, but anticonvulsant drugs, clonazepam, and botulinum toxin injections were the most beneficial. Awareness of the variable phenomenology and the spectrum of causes associated with secondary PxD will allow for more timely diagnosis and early intervention. © 2002 Movement Disorder Society</div></front></TEI><affiliations><list><country><li>États-Unis</li></country><region><li>Texas</li></region><settlement><li>Houston</li></settlement><orgName><li>Baylor College of Medicine</li></orgName></list><tree><country name="États-Unis"><region name="Texas"><name sortKey="Blakeley, Jaishri" sort="Blakeley, Jaishri" uniqKey="Blakeley J" first="Jaishri" last="Blakeley">Jaishri Blakeley</name></region><name sortKey="Jankovic, Joseph" sort="Jankovic, Joseph" uniqKey="Jankovic J" first="Joseph" last="Jankovic">Joseph Jankovic</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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